How to Starve Cancer: The Discovery of a Metabolic Cocktail that could Transform the Lives of Millions

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McLelland underwent surgery to remove the tumor in her lung, and she endured six months of chemo (at a much lower dose than that recommended by her oncologist). But this time, she also employed a strategy to starve the cancer’s stem cells.

Years later, she also discovered the anti-cancer effects of the antibiotic doxycycline (which slows the creation of new cancer cells) and of the anti-worming drug mebendazole (which stops the cancer cells from being able to take on more glucose). McLelland was reluctant about writing her book, but she felt she had a duty to share with the world what she had discovered—and what had saved her life. McLelland had repeatedly been told that diet had no impact on cancer, but the fact that her lung tumor had remained the size of a golf ball—and the fact that there were no tumors in other locations in her body—proved to her that the dietary changes she had already made were making a difference.One man who followed her protocol took his PSA numbers from 1008 down to .67. She’s also helped a stage IV pancreatic cancer patient achieve full remission—another success story unheard of in the medical world. I couldn’t understand why I was controlling one cancer without controlling the other,” said McLelland. “But it’s all about metabolism. The metabolism of my leukemia was totally different from that of my cervical cancer. So, with my low glycemic index diet, I was controlling the cervical cancer, but I wasn’t controlling the leukemia, which instead thrives on proteins.”

One fan even asked for more updates regarding his lifestyle change and wrote: "Ooh could you post more of your meal pics? Very interested on how this works and what you eat, amazing. Keep on keeping on". I already knew that statins would be potentially useful against cervical cancer. But research had also shown that they caused apoptosis in acute myeloid leukemias. I also had overlooked the fact that NSAIDs could cause cell death (apoptosis),” said McLelland. “What I learned from the Life Extension article was that there was a synergy between the two drugs, making them far more potent when taken together.” Unfortunately, just a few months later, Jane started coughing up blood, and found out that her worst nightmare had come true: Her cervical cancer had spread to her lungs. STARS! I'm in tears. I just finished chapter 19 of this book. I just love Jane! I love her. Her ❤️, her tenacity in the face of crazy levels of difficulty, and her desire to share!!

According to doctors and cancer statistics, she should have lived only about 12 weeks after receiving her diagnosis of stage IV cancer.

Suggesting that people fast or starve themselves to kill a tumor has been the domain of dubious and exaggerated claims over the years, and that is not the suggestion now. In recent trials, metabolic pathways have been targeted though various approaches to changing what people eat. Some research has involved minimizing sugar intake. Indeed, some cancer cells metabolize glucose at higher than normal levels (to support the process of aerobic glycolysis), and depleting their access to sugar can slow growth. While the sugar-and-insulin angle has shown promise, more of the research has focused on dietary protein—or, specifically, individual amino acids that make up that protein. Studies have shown that the restriction of the amino acids serine and glycine can modulate cancer outcomes. According to a 2018 study in Nature, the chemotherapy drug methotrexate is affected by the amino acid histidine. Another, asparagine, is involved in the progression of breast cancer metastasis. And a breast cancer patient who was told by her oncologist she was going to die is still alive and well, going to the gym, working as a nurse, and living a full life—all as a result of following McLelland’s approach to starving her cancer. After intensive research, McLelland concluded that all of these drugs would starve the cancer from different angles: dipyridamole cut off cancer’s access to protein, metformin cut off access to glucose, and the statin cut off access to fat. Once the cancer cells were in their weakened state, the addition of etodolac could help finish them off. I didn’t want to have to relive everything,” said McLelland, “but I knew I had information I had to pass on. It was a social responsibility to provide people with information they weren’t getting elsewhere.”However, McLelland cautions that there is no one-size-fits-all approach. Her own experience with her various forms of cancer highlight that fact. Instead, McLelland’s approach focuses on learning which fuel sources your particular cancer uses—and then creating a targeted treatment plan based on that information. The importance of nutrition has long been accepted for conditions such as diabetes and hypertension, diagnoses that come with well-known dietary prescriptions. Even the most commonly used drug in type 2 diabetes, metformin, has been found in clinical trials to be inferior to diet and exercise. Cell biologists like Locasale see extending that line of thinking to cancer as a logical step, because at the cellular level, cancer is also a disease of metabolic pathways. My mother’s cancer was a huge wake-up call to me to re-evaluate the situation I’d found myself in. For the first time ever, I realized I was only one step away from terminal cancer,” McLelland said. “That’s when I started looking at diet and supplementation in more detail as a way to combat cancer.” That’s, uh, yeah,” he attempted to patiently explain. “It’s basically saying we can quantify what’s happening in the cells.”